Poole, David C., Burnley, Mark, Vanhatalo, Anni, Rossiter, Harry B., Jones, Andrew M. (2016) Critical Power: An Important Fatigue Threshold in Exercise Physiology. Medicine and Science in Sports and Exercise, 48 (11). pp. 2320-2334. ISSN 0195-9131. E-ISSN 1530-0315. (doi:10.1249/MSS.0000000000000939) (KAR id:54771)
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Official URL: http://dx.doi.org/10.1249/MSS.0000000000000939 |
Abstract
The hyperbolic form of the power-duration relationship is rigorous and highly conserved across species, forms of exercise and individual muscles/muscle groups. For modalities such as cycling, the relationship resolves to two parameters, the asymptote for power (critical power, CP) and the so-called W' (work doable above CP), which together predict the tolerable duration of exercise above CP. Crucially, the CP concept integrates sentinel physiological profiles - respiratory, metabolic and contractile - within a coherent framework that has great scientific and practical utility. Rather than calibrating equivalent exercise intensities relative to metabolically distant parameters such as the lactate threshold or V[spacing dot above]O2 max, setting the exercise intensity relative to CP unifies the profile of systemic and intramuscular responses and, if greater than CP, predicts the tolerable duration of exercise until W' is expended, V[spacing dot above]O2 max is attained, and intolerance is manifested. CP may be regarded as a 'fatigue threshold' in the sense that it separates exercise intensity domains within which the physiological responses to exercise can (<CP) or cannot (>CP) be stabilized. The CP concept therefore enables important insights into 1) the principal loci of fatigue development (central vs. peripheral) at different intensities of exercise, and 2) mechanisms of cardiovascular and metabolic control and their modulation by factors such as O2 delivery. Practically, the CP concept has great potential application in optimizing athletic training programs and performance as well as improving the life quality for individuals enduring chronic disease.
Item Type: | Article |
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DOI/Identification number: | 10.1249/MSS.0000000000000939 |
Uncontrolled keywords: | exercise intolerance; pulmonary gas exchange; blood lactate; muscle metabolites; vascular control; hypoxia; hyperoxia; heart failure; COPD; disease; aging |
Subjects: |
Q Science Q Science > QP Physiology (Living systems) |
Divisions: | Divisions > Division of Natural Sciences > Sport and Exercise Sciences |
Depositing User: | Mark Burnley |
Date Deposited: | 01 Apr 2016 14:44 UTC |
Last Modified: | 05 Nov 2024 10:43 UTC |
Resource URI: | https://kar.kent.ac.uk/id/eprint/54771 (The current URI for this page, for reference purposes) |
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